This workshop will address the application of available wearable devices to novel uses beyond simple seizure detection and alerting and discuss ongoing research to leverage available technology to better understand seizure risk,aid in epilepsy diagnosis and dynamically assess disease comorbidities.
There are now several options available for treating refractory epilepsy with neurostimulation. It can be difficult to decide when to use each one. We will briefly present a case, followed by presentations on the multiple clinically available neurostimulation modalities: responsive neurostimulation, deep brain stimulation, and vagus nerve stimulation. There will then be a presentation on what practicing neurologists should know about the FDA device clearance process. Then there will be a short talk on choosing a neurostimulation modality in 2019, followed by a panel discussion relating to the presented case,
Translational Research Symposium: Advances in Translation for Traumatic Brain Injury (TBI) and Antiepileptogenesis
This symposium focuses on the current state of research on Traumatic Brain Injury (TBI) and Antiepileptogenesis with particular emphasis on the challenges, achievements and possible solutions for translating ideas into therapies. This sesion will provide an overview of the challenges of translating basic findings to clinical utility in this specific domain of epilepsy research with speakers giving up-to-date reviews of the state of knowledge from both animal and clinical studies; a debate format discussion of the utility of animal work in pre-clinical research; an update on EpiBioS4Rx; a discussion of
The Annual Course will tackle the state-of-the-art knowledge and thought processes related to the growing appreciation of epilepsy as a disorder of brain networks. Through a series of case presentations, didactic lectures, and debates, thought leaders will discuss diagnostic methods and management strategies that address epilepsy and its co-morbidities.
This workshop will address novel circuit therapies in epilepsy. Conventional systemic antiepileptic drug therapy immerses the entire brain, affecting both normal and pathogenic circuits, causing both therapeutic and negative outcomes. In addition, the neuronal circuit disruptions underlying phenotypic expression in epilepsy are mechanistically complex. Targeting individual symptoms has proven insufficiently effective in ameliorating seizures or restoring cognitive function in chronic epilepsy. Presentations in this workshop highlight that altered excitability in specific circuits is a proximate generator of both seizures and aberrant behavior in epilepsy, and that targeting these alterations in